Asthma causation and the gastrointestinal microbiome and metabolome: Might there be a signal, or is it just noise?

Richard Hansen; Kostas Gerasimidis; Steve Turner

doi:10.1016/j.jaci.2019.04.026

Asthma causation and the gastrointestinal microbiome and metabolome: Might there be a signal, or is it just noise?

Richard Hansen, Kostas Gerasimidis, Steve Turner^* (Corresponding Author)

^*Corresponding author for this work

Research output: Contribution to journal › Editorial › peer-review

5 Citations (Scopus)

Abstract

The respiratory tract and gastrointestinal tract (GIT) share common embryologic origins, and an intriguing and commonly revisited question is the following: “Do processes occurring somewhere along the length of the GIT cause asthma symptoms?” Gastroenterologists have been known to remind their pulmonology colleagues that the lungs are actually the larger of 2 appendices of the GIT. Might it be feasible that processes taking place in the GIT can cause respiratory symptoms?

Original language	English
Pages (from-to)	401-403
Number of pages	3
Journal	Journal of Allergy and Clinical Immunology
Volume	144
Issue number	2
Early online date	10 May 2019
DOIs	https://doi.org/10.1016/j.jaci.2019.04.026
Publication status	Published - Aug 2019

Keywords

Asthma
child
gastrointestinal tract
metabolome
microbiome

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title = "Asthma causation and the gastrointestinal microbiome and metabolome: Might there be a signal, or is it just noise?",

abstract = "The respiratory tract and gastrointestinal tract (GIT) share common embryologic origins, and an intriguing and commonly revisited question is the following: “Do processes occurring somewhere along the length of the GIT cause asthma symptoms?” Gastroenterologists have been known to remind their pulmonology colleagues that the lungs are actually the larger of 2 appendices of the GIT. Might it be feasible that processes taking place in the GIT can cause respiratory symptoms?",

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KW - child

KW - gastrointestinal tract

KW - metabolome

KW - microbiome

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Asthma causation and the gastrointestinal microbiome and metabolome: Might there be a signal, or is it just noise?

Abstract

Keywords

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