System-Based Solutions to Minimizing Nephrotoxin-Induced Acute Kidney Injury

Matthew  James; Simon Sawhney

doi:10.2215/CJN.0000000000000053

System-Based Solutions to Minimizing Nephrotoxin-Induced Acute Kidney Injury

Matthew James, Simon Sawhney

University of Calgary

Research output: Contribution to journal › Editorial › peer-review

Abstract

Drug-associated AKI is a common cause of iatrogenic AKI in hospitalized patients. Drugs can cause AKI via a variety of mechanisms, including parenchymal damage to the kidneys by acute tubular injury, acute interstitial nephritis, or intratubular precipitation of crystals, as well as through hemodynamic effects that can lower glomerular filtration rate without causing parenchymal injury.

Original language	English
Pages (from-to)	157-159
Journal	Clinical Journal of the American Society of Nephrology
Volume	18
Issue number	2
DOIs	https://doi.org/10.2215/CJN.0000000000000053
Publication status	Published - Feb 2023

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abstract = "Drug-associated AKI is a common cause of iatrogenic AKI in hospitalized patients. Drugs can cause AKI via a variety of mechanisms, including parenchymal damage to the kidneys by acute tubular injury, acute interstitial nephritis, or intratubular precipitation of crystals, as well as through hemodynamic effects that can lower glomerular filtration rate without causing parenchymal injury. ",

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AB - Drug-associated AKI is a common cause of iatrogenic AKI in hospitalized patients. Drugs can cause AKI via a variety of mechanisms, including parenchymal damage to the kidneys by acute tubular injury, acute interstitial nephritis, or intratubular precipitation of crystals, as well as through hemodynamic effects that can lower glomerular filtration rate without causing parenchymal injury.

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JO - Clinical Journal of the American Society of Nephrology

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System-Based Solutions to Minimizing Nephrotoxin-Induced Acute Kidney Injury

Abstract

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